Specimen and client information
Specimen type
Buccal (cheek) swab
Date reported
29 July 2026
The reports inside this package
This one document contains 6 complete genetic reports. Each was scored on its own, then combined here so you can read everything in one place. Every result further down is labelled with the report it came from.
Longevity & Healthy Aging29 results reported of 33 analytes · 50 recommendations
Mitochondrial Support28 results reported of 28 analytes · 50 recommendations
Bone Health17 results reported of 17 analytes · 50 recommendations
Joint & Tendon Health27 results reported of 27 analytes · 50 recommendations
Inflammation & Autoimmunity80 results reported of 83 analytes · 50 recommendations
Toxins & Detox55 results reported of 58 analytes · 50 recommendations
About this report
This report reads your DNA and sorts every result into one of five plain-language groups, so you can see at a glance what deserves attention and what does not. Nothing here is a diagnosis. A flagged result means your genetics place you above or below the average person for that trait — a reason to ask a question, not a reason to worry.
What each result group means
Worth a conversation with your doctor
These came back with a higher genetic predisposition than most people. That is not a diagnosis — it means these are the ones worth raising at your next appointment.
Running above the typical range
Your genetics point to these sitting higher than average. Often useful to know, sometimes worth monitoring.
Running below the typical range
Your genetics point to these sitting lower than average. Same idea — context, not alarm.
Working in your favour
Good news. Your genetics look protective or better than average here.
In line with most people
Nothing unusual. These were tested and came back typical — worth having on record.
Your results at a glance
How to read this. A result being flagged does not mean you have a condition — it means your genetics put you above or below the average person for that trait. Of the 206 results here, 30 came back worth discussing with a clinician and 17 came back in your favour. Bring this document to your appointment.
What this package looks at
Longevity & Healthy Aging — Longevity is a fascinating and complex area of study that encompasses not only the length of life but also the quality of life as we age. Genetic factors play a significant role in determining how long we live and how well we age.
Mitochondrial Support — Think of mitochondria as tiny power plants inside each of your cells, converting the food you eat into energy that keeps you alive and thriving. But these remarkable cellular structures do much more than just produce energy -they're key players in your overall health and vitality.
Bone Health — Bone health is crucial for maintaining a strong skeletal system, mobility, and overall physical well-being. While lifestyle factors such as diet and exercise play important roles in bone health, genetics also significantly influences how strong and resilient your bones are.
Joint & Tendon Health — Joint and tendon health is essential for maintaining mobility, flexibility, and overall physical function. Genetic factors play a significant role in determining your susceptibility to various conditions that affect the joints and tendons, such as joint pain, arthritis, and tendon problems. While
Inflammation & Autoimmunity — Inflammation and autoimmunity are key processes that affect the body’s ability to maintain health and respond to threats. Inflammation is the body's natural response to injury or infection, but chronic inflammation can lead to a variety of health problems, from gut issues to systemic diseases.
Toxins & Detox — The impact of environmental toxins on health is an increasingly important topic in personalized health and functional medicine. Our genetic makeup plays a significant role in how our bodies process, detoxify, and respond to various toxins.
What you can actually do
These are ordered by how closely they relate to your flagged results — not generic health advice. Marrow does not prescribe, dose, or tell you to take anything. Diet and lifestyle changes are listed first because they are yours to make. Anything you would swallow is listed separately, by name only, as something to raise with a clinician who knows your history and your medications.
Changes you can make yourself
1
Manual TherapyReduce Tendon Strain To reduce tendon strain, modify your daily activities to avoid motions that cause discomfort or pain.
Relates to: Shoulder Impingement, Jaw Disorders (TMJ)
2
Mediterranean DietLimit red meat to a few times a month and include a moderate amount of dairy products.
Relates to: Artery Hardening, Frailty, PPARD (Longevity), Prostate Cancer
3
Physical Therapy30 minutesAttend physical therapy sessions 2-3 times per week for a duration of 4-6 weeks, depending on your specific TA condition and the advice of your healthcare provider.
Relates to: Shoulder Impingement, Jaw Disorders (TMJ), Neck Arthritis
4
Whole-Food Plant-Based Dietfew weeks to avoid digestive discomfort.
Relates to: Pancreas Inflammation, Eye Inflammation, Myocarditis
5
Shock Wave TherapySchedule and attend sessions for extracorporeal shock wave therapy (ESWT) with a qualified healthcare provider.
Relates to: Heel Pain (Plantar Fasciitis), Shoulder Impingement, Trigger Finger
6
Strength TrainingEngage in strength training exercises, such as weight lifting or bodyweight exercises, for 60 minutes per session, TA 2 to 3 times per week.
Relates to: Colorectal Cancer, Artery Hardening, PPARD (Longevity), Prostate Cancer
7
Exercise TherapyBegin by consulting with a physical therapist to assess your condition and create a personalized exercise plan.
Relates to: Shoulder Impingement, Jaw Disorders (TMJ)
8
Reduce Jaw StrainAvoid chewing gum, biting on hard foods like nuts or ice, and lengthy yawning.
Relates to: Jaw Disorders (TMJ)
9
Eat Fiber-Rich Foodsgrams per day, spread out over all meals.
Relates to: Colorectal Cancer, Artery Hardening
Worth asking your clinician about
These came up in the research behind your flagged results. They are listed by name so you can ask about them. Marrow is not recommending them, and gives no dose. Supplements interact with prescription medication and with each other — that conversation belongs with your clinician.
1
Maintain Optimal Vitamin D LevelsRelates to: Calcium, Hypoparathyroidism, Crohn’s Disease, Thyroid Inflammation
2
Glutathione supplementsRelates to: UGT2A1 (Cognition)
3
N-acetylcysteine (NAC)Relates to: UGT2A1 (Cognition), Aluminum
4
Green TeaRelates to: Prostate Cancer, Artery Hardening, Colorectal Cancer, Aluminum
5
Omega-3 (Fish Oil)Relates to: SH2B3 (Autoimmunity), Crohn’s Disease, Pancreas Inflammation, Psoriasis
Your headline findings
Of the 206 results in this package, these 8 came back furthest from the population average. Each one below explains what it is, where you sit, and which report it came from. Start here.
Heel Pain (Plantar Fasciitis)More likely
More likely to have plantar fasciitis based on 28,598 genetic variants we looked at
Your risk is greater than 99% of the population and lower than 1% of the population.
What this actually is
Factors that might increase the risk of developing plantar fasciitis include: Age, with it being most common between the ages of 40 and 60 Certain types of exercise, such as long-distance running or ballet dancing Foot mechanics, including having a high arch, flat feet, or an abnormal walking pattern Obesity, which places extra stress on the plantar fascia Occupations that require long periods of standing or walking on hard surfaces Wearing shoes with inadequate support Genetics While plantar fasciitis is primarily associated with environmental factors and physical stress on the foot, there is some evidence to suggest that genetic factors might play a role in predisposing certain individuals to the condition.
What you can do
- Shock Wave Therapy
Schedule and attend sessions for extracorporeal shock wave therapy (ESWT) with a qualified healthcare provider. - Low-Level Laser Therapy (LLLT)
Use a low-level laser therapy device, as directed by its manual or a healthcare professional, on the affected area. - Acupuncture
Visit a licensed acupuncturist for a session, typically lasting between 30 to 60 minutes, once or twice a week.
From your Joint & Tendon Health report
Tonsil InflammationMore likely
Higher predisposition than 99% of people
Your risk is greater than 99% of the population and lower than 1% of the population.
What this actually is
The course of cause is viral or require specific through pain Bacterial can require Recurrent cases of recommendation for of the tonsils. are also common cause.
Ask your clinician about
- Maintain Optimal Vitamin D Levels
Named only. Marrow gives no dose and does not recommend these.
From your Inflammation & Autoimmunity report
Neck ArthritisMore likely
Higher predisposition than 98% of people
Your result sits higher than 98% of the population.
What this actually is
As the disks in the cervical spine dehydrate and shrink, signs of osteoarthritis develop, including the formation of bony projections along the edges of bones (bone spurs).
Cervical spondylosis can sometimes lead to a narrowing of the space needed by the spinal cord and the nerve roots that pass through the spine to the rest of the body.
More likely to have When these nerve pathways are affected, individuals might 481,932 genetic experience neurological symptoms such as numbness, weakness, or tingling in the arms, hands, legs, or feet.
What you can do
- Physical Therapy 30 minutes
Attend physical therapy sessions 2-3 times per week for a duration of 4-6 weeks, depending on your specific TA condition and the advice of your healthcare provider. - Acupuncture
Visit a licensed acupuncturist for a session, typically lasting between 30 to 60 minutes, once or twice a week. - Topical CBD
Apply a CBD-infused cream or ointment to the affected area of the skin 2-3 times per day, gently massaging it into the skin until fully absorbed.
From your Joint & Tendon Health report
Excessive Mast CellsMore likely
More likely to have excessive mast cells based on 14 genetic variants we looked at
Your risk is greater than 97% of the population and lower than 3% of the population.
What this actually is
Mastocytosis is often linked to mutations in the KIT gene, which plays a role in mast cell growth and development.
These mutations are usually acquired during a person's life rather than being inherited.
Other, less important risk factors include: Family history: Rare cases may run in families, but most are sporadic.
What you can do
- Avoid Exposure to Molds
Keep indoor humidity levels below 50% by using dehumidifiers or air conditioners, ensure proper ventilation in areas like bathrooms and kitchens, fix leaks promptly, and clean up any water damage within 24-48 hours to prevent mold growth.
Ask your clinician about
Named only. Marrow gives no dose and does not recommend these.
From your Mitochondrial Support report
Thyroid InflammationMore likely
More likely to have thyroid inflammation based on 348,866 genetic variants we looked at
Your risk is greater than 96% of the population and lower than 4% of the population.
What this actually is
The most common forms of thyroiditis include Hashimoto's thyroiditis, an autoimmune disease where the body's immune system mistakenly attacks the thyroid; postpartum thyroiditis, which can occur after childbirth; and subacute thyroiditis, which usually follows a viral infection.
Diagnosis may involve blood tests to measure thyroid hormone levels, as well as antibody tests to detect autoimmune activity.
Treatment depends on the type and severity of thyroiditis, ranging from observation and symptom management to medications like levothyroxine for hypothyroidism or beta-blockers for hyperthyroidism symptoms.
What you can do
- Autoimmune Protocol (AIP) Diet
Eliminate grains, legumes, nuts, seeds, nightshades, dairy, eggs, coffee, alcohol, refined sugars, refined oils, food additives, and processed foods from your diet for a minimum of 30 days.
Ask your clinician about
- Maintain Optimal Vitamin D Levels
Named only. Marrow gives no dose and does not recommend these.
From your Inflammation & Autoimmunity report
Artery HardeningMore likely
More likely to have atherosclerosis based on 51,588 genetic variants we looked at
Your risk is greater than 95% of the population and lower than 5% of the population.
What this actually is
Key Takeaways: About 60% of differences in people's chances of having atherosclerosis may be due to genetics Risk factors: being male, high blood pressure and cholesterol, diabetes, obesity, smoking, lack of exercise, poor diet, and age.
Artery hardening begins at a young age and your genetics, lifestyle, diet, and exercise may all impact how much and how fast it develops over the course of your lifetime.
The arteries are blood vessels that carry oxygen and nutrients from the heart to the rest of the body.
What you can do
- Mediterranean Diet
Limit red meat to a few times a month and include a moderate amount of dairy products. - Strength Training
Engage in strength training exercises, such as weight lifting or bodyweight exercises, for 60 minutes per session, TA 2 to 3 times per week. - Eat Fiber-Rich Foods
grams per day, spread out over all meals.
From your Longevity & Healthy Aging report
Pancreas InflammationMore likely
More likely to get pancreas inflammation based on 1,669 genetic variants we looked at
Your risk is greater than 95% of the population and lower than 5% of the population.
What this actually is
The pancreas is an organ located behind the stomach that releases crucial enzymes for carbs and fats digestion.
Pancreatitis is inflammation of the pancreas, which can be acute or chronic.
Potential risk factors for pancreatitis include [R, R, R]: Alcohol Cigarette smoking Obesity High blood lipids Certain medications Genetics Genetically predicted higher fasting insulin may be associated with acute and chronic pancreas inflammation.
What you can do
- Whole-Food Plant-Based Diet
few weeks to avoid digestive discomfort.
Ask your clinician about
- Probiotics
- Omega-3 (Fish Oil)
Named only. Marrow gives no dose and does not recommend these.
From your Inflammation & Autoimmunity report
Prostate CancerMore likely
More likely to get prostate cancer based on 1,049,413 genetic variants we looked at
Your risk is greater than 94% of the population and lower than 6% of the population.
What this actually is
The exact cause of prostate cancer is not clearly understood, but several factors have been identified that increase the risk of developing this disease: Age: The risk increases significantly after age 50, and it is most common in men over 65.
Family history: Having a father or brother with prostate cancer more than doubles a man’s risk.
Race/Ethnicity: African-American men have a higher risk of prostate cancer than men of other races.
What you can do
- Mediterranean Diet
Limit red meat to a few times a month and include a moderate amount of dairy products.
Ask your clinician about
- Green Tea
- Green Tea Extract
Named only. Marrow gives no dose and does not recommend these.
From your Longevity & Healthy Aging report
A further 22 results were also flagged for attention. They are listed below and explained in full in the technical version of this report.
Where your genetics are working for you
Not everything in a genetic report is a warning. These came back protective or better than average.
Cognitive DeclineLess likely
Less likely to have cognitive decline based on 272,168 genetic variants we looked at
Your risk is greater than 1% of the population and lower than 99% of the population.
What this actually is
Mild cognitive decline is a normal part of aging that can affect cognitive functions such as memory, attention, and problem-solving.
About 60-70% of the differences in people’s cognitive decline may come from genetics.
For example, genetically high total and bioavailable testosterone may be causally associated with larger gray matter volume in men [R, R, R].
From your Longevity & Healthy Aging report
Joint PainLess likely
Less likely to have osteoarthritis based on 385,825 genetic variants we looked at
Your risk is greater than 1% of the population and lower than 99% of the population.
What this actually is
Key Takeaways: Osteoarthritis will affect the majority of people over the age of 55.
So, even with low genetic risk, the overall risk is still high for older people.
About 50% of the differences in people's chances of getting osteoarthritis may be due to genetics.
From your Joint & Tendon Health report
Bone SpursLess likely
Less likely to have bone spurs based on 1,676 genetic variants we looked at
Your risk is greater than 5% of the population and lower than 95% of the population.
What this actually is
Bone spurs develop as a result of bone and joint degeneration and are primarily associated with the aging process.
However, they can also arise from underlying diseases and conditions that affect bone and joint health: Osteoarthritis: The most common cause of bone spurs, osteoarthritis involves the breakdown of cartilage, leading to joint damage and increased bone friction.
Spinal degeneration: Bone spurs commonly occur in the spine as a result of the degeneration of discs and joints, often due to aging.
From your Bone Health report
Everything else that came back
The remaining results, grouped by what they mean. Every one of these is explained in full — with the genes and variants behind it — in the technical version of this report.
Worth a conversation with your doctor (22)
- Shoulder Impingement — More likely, 90th percentile
- Disc Degeneration — More likely, 90th percentile
- Frailty — More likely, 89th percentile
- Urethra Inflammation — More likely, 88th percentile
- Trigger Finger — More likely, 87th percentile
- Eye Inflammation — More likely, 84th percentile
- Jaw Disorders (TMJ) — More likely, 81th percentile
- Dupuytren’s Contracture — More likely, 80th percentile
- Colorectal Cancer — More likely, 73th percentile
- Crohn’s Disease — More likely, 66th percentile
- Psoriasis — More likely, 65th percentile
- PPARD (Longevity) — Worse
- Mitochondrial Health (Functional) — Worse
- Bone Cancer — More likely
- Hypoparathyroidism — More likely
- Calcium — Increased need
- Vitamin K — Increased need
- Spinal Arthritis — More likely
- Myocarditis — More likely
- Nephritis — More likely
- ITGB3 (Mold Sensitivity) — Worse genetics
- SOD3 (Oxidative Stress) — Worse
Running above the typical range (23)
- Aluminum — Higher levels, 83th percentile
- HLA (Inflammation) — Higher activity, 80th percentile
- CFH (Eye Health/ Longevity) — Higher activity
- Fluoride Sensitivity — Higher
- NQO1 (Detox) — Higher activity
- GPX1 (Glutathione/Detox) — Higher activity
- SUOX (PCOS/ Blood Sugar) — Higher activity
- GC (Vitamin D) — Higher activity
- MMP1 (Collagen) — Higher activity
- Basophils — Higher levels
- HLA-DOB (Inflammation) — Higher activity
- NF-kB (Inflammation) — Higher activity
- NLRP3 (Gut Inflammation) — Higher activity
- ANKRD55 (Autoimmune) — Higher activity
- ABCB1 (Chronic Lyme) — Higher activity
- HNMT (Histamine) — Higher activity
- PBDE Sensitivity (CYP2B6) — Higher
- CYP1B1 (Detox/ Skin Health) — Higher activity
- SULT1A1 (Detox) — Higher activity
- GSTP1 (Detox) — Higher activity
- Sulfation (Detox) — Higher activity
- UGT (Detox) — Higher activity
- CYP1A2 (Detox) — Higher activity
Running below the typical range (18)
- ADA (Cognition/Longevity) — Lower activity
- CETP (Cholesterol/ Longevity) — Lower activity
- SOD2 (Oxidative Stress) — Lower activity
- SH2B3 (Autoimmunity) — Lower activity
- HNF4A (Gut Inflammation) — Lower activity
- CTLA4 (Autoimmunity) — Lower activity
- IL1B (Inflammation/ Fatigue) — Lower activity
- JAK2 (Gut Inflammation) — Lower activity
- IL8 (Inflammation) — Lower activity
- IL21 (Autoimmunity & Allergies) — Lower activity
- CHIT1 (Mold Sensitivity) — Lower activity
- GSTA1 (Detox/ Glutathione) — Lower activity
- PAH Sensitivity — Lower
- PON1 (Detox) — Lower activity
- UGT2A1 (Cognition) — Lower activity
- GCLC (Glutathione & Detox) — Lower activity
- SLCO1B1 (Detox) — Lower activity
- COQ2 (Oxidative Stress) — Lower activity
Working in your favour (14)
- Scoliosis — Less likely, 19th percentile
- Tendon Inflammation — Less likely, 19th percentile
- Optic Nerve Inflammation — Less likely, 19th percentile
- Tendon Injury — Less likely, 18th percentile
- Gut Inflammation — Less likely, 8th percentile
- Appendicitis — Less likely, 8th percentile
- Muscle Loss — Less likely
- Lung Cancer — Less likely
- IGF1R (Longevity) — Better
- Ehlers-Danlos Syndrome (Progeroid — Less likely
- ADAMTS5 (Joint Health) — Better genetics
- Diverticular Disease — Less likely
- Sjogren's Syndrome — Less likely
- IL6R (Weight) — Better genetics
In line with most people (118)
- Achilles Tendon Injury — Typical likelihood, 79th percentile
- Aging Eyes — Typical likelihood, 77th percentile
- DNA Damage — Typical levels, 66th percentile
- Alzheimer's Disease — Typical likelihood, 65th percentile
- Bone Infection — Typical likelihood, 65th percentile
- Hip Pain — Typical likelihood, 64th percentile
- Ulcerative Colitis — Typical likelihood, 63th percentile
- Cardiovascular Disease — Typical likelihood, 60th percentile
- Spinal Canal Narrowing — Typical likelihood, 60th percentile
- Multiple Sclerosis — Typical likelihood, 60th percentile
- Gout — Typical likelihood, 58th percentile
- Cadmium — Typical levels, 57th percentile
- Carpal Tunnel Syndrome — Typical likelihood, 56th percentile
- Age-Related Macular Degeneration — Typical likelihood, 44th percentile
- Stomach Inflammation — Typical likelihood, 42th percentile
- Celiac Disease — Typical likelihood, 42th percentile
- Rheumatoid Arthritis — Typical likelihood, 40th percentile
- Joint Stiffness — Typical likelihood, 39th percentile
- HRH4 (Allergies, Inflammation) — Typical activity, 39th percentile
- Fractures — Typical likelihood, 37th percentile
- Inflammation (CRP) — Typical levels, 36th percentile
- Knee Pain — Typical likelihood, 36th percentile
- Tendon Lining Inflammation — Typical likelihood, 36th percentile
- Osteoporosis — Typical likelihood, 33th percentile
- Eczema — Typical likelihood, 29th percentile
- Lead — Typical levels, 28th percentile
- Organophosphate Sensitivity — Typical, 21th percentile
- IL-6 — Typical levels, 20th percentile
- Pesticide Sensitivity — Typical, 18th percentile
- Arsenic — Typical levels, 16th percentile
- Type 1 Diabetes — Typical likelihood, 15th percentile
- Lupus — Typical likelihood, 12th percentile
- Facial Wrinkles — Typical
- Longevity — Typical
- Healthy Aging — Typical
- Hair Graying — Typical
- Pancreatic Cancer — Typical likelihood
- SIRT1 (Longevity) — Typical activity
- TERT (Longevity) — Typical activity
- OBFC1 (Longevity) — Typical activity
- AKT1 (Longevity) — Typical genetics
- APOC3 (Blood Lipids/ Longevity) — Typical activity
- FOXO3 (Longevity) — Typical activity
- TAS2R16 (Bitter Taste/ Longevity) — Typical activity
- PARP1 (Longevity, Cognition) — Typical genetics
- Detox — Typical ability
- Oxidative Stress — Typical
- Benzene Sensitivity — Typical
- NFE2L2/NRF2 (Detox) — Typical activity
- Sensitivity to Airborne Mold — Typical
- Sensitivity to Foodborne Mold — Typical
- Multiple Chemical Sensitivity — Typical likelihood
- BPA Sensitivity — Typical
- Mercury — Typical levels
- Phase I Detox — Typical ability
- IgE — Typical levels
- IL-10 — Typical levels
- IL6 Gene (Inflammation) — Typical activity
- Cold Intolerance — Typical likelihood
- PPARGC1A (Fitness/Blood Sugar) — Typical activity
- TNF Gene (Inflammation) — Typical activity
- Hyperparathyroidism — Typical likelihood
- Vitamin D — Typical need
- Phosphate — Typical levels
- Magnesium — Typical need
- VDR (Vitamin D) — Typical activity
- CYP2R1 (Vitamin D) — Typical activity
- Psoriatic Arthritis — Typical likelihood
- Rheumatic Fever — Typical likelihood
- Ankylosing Spondylitis — Typical likelihood
- COL5A1 (Collagen) — Typical activity
- Chronic Lyme — Typical likelihood
- Sepsis — Typical likelihood
- Pulmonary Fibrosis — Typical likelihood
- Actinic Keratosis — Typical likelihood
- Meningitis — Typical likelihood
- Sarcoidosis — Typical likelihood
- Vitiligo — Typical likelihood
- Alopecia Areata — Typical likelihood
- Systemic Sclerosis — Typical likelihood
- Pemphigus Vulgaris — Typical likelihood
- Behcet’s Disease — Typical likelihood
- IgA Nephropathy — Typical likelihood
- TNF — Typical levels
- White Blood Cells — Typical levels
- Eosinophils — Typical levels
- Monocytes — Typical levels
- Neutrophils — Typical levels
- Erythrocyte Sedimentation Rate — Typical levels
- IL-17 (Th17) — Typical levels
- HLA-B (Autoimmune) — Typical
- HLA-DQ (Inflammation) — Typical activity
- HLA-DRB1 (Autoimmunity) — Typical
- HLA-DQA2 (Inflammation) — Typical
- TLR4 (Inflammation) — Typical activity
- PTPN22 (Autoimmunity) — Typical
- DAO (Histamine) — Typical activity
- STAT3 (Th1/Th17) — Typical activity
- IL13 (Allergies, Lung Function) — Typical activity
- IL4 (Allergies, Autoimmunity) — Typical activity
- CRP Gene — Typical activity
- PADI4 (Autoimmune) — Typical activity
- IL10 Gene (Autoimmunity) — Typical activity
- SLC22A4 (Gut Inflammation) — Typical genetics
- Glyphosate Sensitivity — Typical
- Air Pollution Sensitivity — Typical
- Phthalate Sensitivity — Typical
- GSDMB (Mold Sensitivity) — Typical activity
- XPC (DNA Damage) — Typical activity
- XRCC (DNA Damage) — Typical activity
- Glutathione — Typical function
- UGT1A1 (Detox) — Typical activity
- AHR (Detox) — Typical activity
- Phase II Detox — Typical ability
- CYP1A1 (Detox) — Typical activity
- NAT2 (Detox) — Intermediate
- Lipid Oxidation — Typical
- GPX4 (Selenium & Glutathione) — Typical activity
What to do with this
Genetics is a predisposition, not a verdict. The single most useful thing you can do with this document is take it to a clinician and talk through the flagged findings — especially any that line up with symptoms you already have or conditions that run in your family. Nothing here changes: your DNA is the same at 60 as it is today, so this report does not expire.
Methodology
Genotyping was performed on DNA extracted from a buccal (cheek) swab using a genome-wide array. Results are derived from peer-reviewed genome-wide association data and, where applicable, published clinical guidelines. Each report was scored independently against its own reference set. Percentile figures describe where your result sits relative to a reference population.
Limitations
This is a laboratory-developed test. It has not been cleared or approved by the U.S. Food and Drug Administration. Genotyping does not detect all variants in the genes assessed; the absence of a flagged result does not exclude risk. Predisposition is probabilistic and is modified by environment, lifestyle, ancestry and family history. Results must be interpreted by a qualified healthcare professional and must not be used alone to diagnose, treat, or alter any medication regimen. Marrow does not recommend, prescribe, or dose any supplement or medication.